Neuronal nitric oxide synthase in the gill of the killifish, Fundulus heteroclitus.

Urotensin II and its receptor in the killifish gill: regulators of NaCl extrusion.

The peptide urotensin II (UII) and its receptor (UT) mediate cardiovascular and renal effects in both mammals and fishes. In both groups, vasopressor and diuretic responses predominate, although, in mammals, some secondary vasodilatation is found, mediated by secondary release of nitric oxide or prostacyclin. In fishes, gill extrusion of NaCl is inhibited by UII, but a single study has determin...

NaCl transport across the opercular epithelium of Fundulus heteroclitus is inhibited by an endothelin to nitric oxide, superoxide, and prostanoid signaling axis

Recent evidence suggests that paracrine signaling agents, such as endothelin (ET), nitric oxide (NO), superoxide (O2 ), and prostanoids can modulate mammalian renal function by affecting both hemodynamic and epithelial, ionic transport pathways. Since these signaling pathways have been described in fish blood vessels, we hypothesized that they may control salt transport across the gill epitheli...

Effects of environmental salinity on gill endothelin receptor expression in the killifish, Fundulus heteroclitus.

We recently determined that rapid changes in environmental salinity alter endothelin-1 (EDN1) mRNA levels in the euryhaline killifish, Fundulus heteroclitus, so we hypothesized that EDN1 may be a local regulator of gill ion transport in teleost fishes. The purpose of the present study was to examine the effects of changes in environmental salinity on the gill endothelin receptors: EDNRA, EDNRB,...

Comparing Methods of Euthanasia and Gill Culture for Hypoxia Research on the Gulf Killifish, Fundulus grandis

NaCl transport across the opercular epithelium of Fundulus heteroclitus is inhibited by an endothelin to NO, superoxide, and prostanoid signaling axis.

Recent evidence suggests that paracrine signaling agents, such as endothelin (ET), nitric oxide (NO), superoxide (O2-), and prostanoids can modulate mammalian renal function by affecting both hemodynamic and epithelial ionic transport pathways. Since these signaling pathways have been described in fish blood vessels, we hypothesized that they may control salt transport across the gill epitheliu...